A CASE OF HYPOKALEMIC PARALYSIS DUE TO A RARE CAUSE SJOGREN SYNDROME
Abstract
53 years, old lady was admitted with sudden onset, weakness of all 4 limbs and neck muscle weakness, she had the similar episodes one month before. Investigations revealed, Nonanion gap metabolic acidosis ANA-, RA factor -negative. Anti SSA antibody Anti SSB antibody positive .ECG shows hypokalemic features, it is a rare case of hypokalemia with primary Sjogren syndrome.
Full Text:
PDFReferences
Teitz Fundemental of clinical biochemistry
Kaplan clinical chemistry
Kellys Rhemotology
Takemoto F, Hoshino J, Sawa N, et al. Autoantibodies against carbonic anhydrase II are increased in renal tubular acidosis associatedwith Sjogren syndrome. Am J Med 2005; 118:181-4.
JooKW,Jeon US, Han JS, et al. Absence of H(+)-ATPasein the intercalatedcells of renal tissues in classic distal renal tubular acidosis.Clin Nephrol 1998; 49: 226-31.
ZimhonyO, Sthoeger Z, Ben David D, et al. Sjogren's syndrome presenting as hypokalemic paralysis due to distal renal tubular acidosis. J Rheumatol 1995; 22: 2366-8.
Rodriguez-Soriano J. New insights into the pathogenesis of renal
tubular acidosis--from functional to molecular studies. PediatricNephrol 2000 ; 14: 1121-36.
Refbacks
- There are currently no refbacks.
This work is licensed under a Creative Commons Attribution-NoDerivatives 4.0 International License.
An Initiative of The Tamil Nadu Dr M.G.R. Medical University